Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Can it be taken: a person having predispositions to any chronic
disease(esp.those having weight loss as promient symptom eg diabetes,
TB etc.) got some symptoms of disease but not yet lost substancial and
unintentional weight, is either in early stage or not really got frank
disease and can be cured by adopting better health practices without
medicines?

Weight loss is a physical finding and not a symptom. The symptom that
leads to weight loss is anorexia which is loss of appetite.

Yes, but can't anorexia be disease mediated/oriented--either for spread
or for cure of disease?

It serves the purpose of letting a physician know that there is a
problem.

In short, whether weght lost symptom is must to declared patient with
frank disease in which weght loss is an important symptom?

Folks with TB lose their appetite. Folks with type 1 diabetes also
lose their appetite. Folkd with type 2 diabetes generally do not lose
their appetite and so they gain weight thereby worsening their
condition.

What about when type2 become IDDM or frank dibetic?

Then the type 2 diabetic typically starts losing his/her appetite.

Do they loose their
appetite if medicated or not medicated?

With loss of homeostasis.

Let us think about TB. Weight loss is an important symptom of active TB

They lose weight from eating less because they have lost their
appetite.

Weght loss may not be there when TB bacterias are in latent stage. I
think, there can be acute/active stage type symptoms when 1.disease is
spreading(direction of progress of disease on wekening of immunity)) 2.
when disease is occasionally controled(direction towards natural cure
by getting strong immunity). But weght loss OR no weight loss/gain
weight can be the differenciating symptom of either disease is toward
spread or toward cure. Right??

See above.

Don't bacterias kill cells resulting weght loss?

No.

Can't anorexia and improvement in appetite be disease mediated/oriented
either for spread or for cure of disease? However loss of appetite be
may also be possible as a direction towards cure for decreasing
nutients/starving the disease causing agents in latencies/toumor?
It is thought provoking to many TB specialists when I discudssed.

Anorexia serves to inform the physician that there is a problem.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld

Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Can it be taken: a person having predispositions to any chronic
disease(esp.those having weight loss as promient symptom eg diabetes,
TB etc.) got some symptoms of disease but not yet lost substancial and
unintentional weight, is either in early stage or not really got frank
disease and can be cured by adopting better health practices without
medicines?

Weight loss is a physical finding and not a symptom. The symptom that
leads to weight loss is anorexia which is loss of appetite.

Yes, but can't anorexia be disease mediated/oriented--either for spread
or for cure of disease?

It serves the purpose of letting a physician know that there is a
problem.

What causes anorexia?

In short, whether weght lost symptom is must to declared patient with
frank disease in which weght loss is an important symptom?

Folks with TB lose their appetite. Folks with type 1 diabetes also
lose their appetite. Folkd with type 2 diabetes generally do not lose
their appetite and so they gain weight thereby worsening their
condition.

What about when type2 become IDDM or frank dibetic?

Then the type 2 diabetic typically starts losing his/her appetite.

It means real disease is matured only when patient start losing his/her
appetite resulting into weight loss?
Do they loose their
appetite if medicated or not medicated?

With loss of homeostasis.

Let us think about TB. Weight loss is an important symptom of active TB

They lose weight from eating less because they have lost their
appetite.

Weght loss may not be there when TB bacterias are in latent stage. I
think, there can be acute/active stage type symptoms when 1.disease is
spreading(direction of progress of disease on wekening of immunity)) 2.
when disease is occasionally controled(direction towards natural cure
by getting strong immunity). But weght loss OR no weight loss/gain
weight can be the differenciating symptom of either disease is toward
spread or toward cure. Right??

See above.

Don't bacterias kill cells resulting weght loss?

No.

Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?
Can't anorexia and improvement in appetite be disease mediated/oriented
either for spread or for cure of disease? However loss of appetite be
may also be possible as a direction towards cure for decreasing
nutients/starving the disease causing agents in latencies/toumor?
It is thought provoking to many TB specialists when I discudssed.

Anorexia serves to inform the physician that there is a problem.

Yes, but can loss or gain of appetite indicate that whether patient is
progressing toward increase in disease or cure of disease respectively?

Do patients, having latent stage of TB, get episodes of acute type
symptoms of TB, when he is naturally improving by gaining immune
strength due to adopting healthful practices? If yes, what will be the
difference in symptoms when acute type symptoms are due to getting cure
or due to progress of disease?

In article om,
"Kumar" wrote:


That's it! You are killfiled along with Chung for crossposting...

Ass hole!!!!

Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Can it be taken: a person having predispositions to any chronic
disease(esp.those having weight loss as promient symptom eg diabetes,
TB etc.) got some symptoms of disease but not yet lost substancial and
unintentional weight, is either in early stage or not really got frank
disease and can be cured by adopting better health practices without
medicines?

Weight loss is a physical finding and not a symptom. The symptom that
leads to weight loss is anorexia which is loss of appetite.

Yes, but can't anorexia be disease mediated/oriented--either for spread
or for cure of disease?

It serves the purpose of letting a physician know that there is a
problem.

What causes anorexia?

The underlying problem that makes a person unwell.

In short, whether weght lost symptom is must to declared patient with
frank disease in which weght loss is an important symptom?

Folks with TB lose their appetite. Folks with type 1 diabetes also
lose their appetite. Folkd with type 2 diabetes generally do not lose
their appetite and so they gain weight thereby worsening their
condition.

What about when type2 become IDDM or frank dibetic?

Then the type 2 diabetic typically starts losing his/her appetite.

It means real disease is matured only when patient start losing his/her
appetite resulting into weight loss?

No. It means there is a loss of homeostasis.

Do they loose their
appetite if medicated or not medicated?

With loss of homeostasis.

Let us think about TB. Weight loss is an important symptom of active TB

They lose weight from eating less because they have lost their
appetite.

Weght loss may not be there when TB bacterias are in latent stage. I
think, there can be acute/active stage type symptoms when 1.disease is
spreading(direction of progress of disease on wekening of immunity)) 2.
when disease is occasionally controled(direction towards natural cure
by getting strong immunity). But weght loss OR no weight loss/gain
weight can be the differenciating symptom of either disease is toward
spread or toward cure. Right??

See above.

Don't bacterias kill cells resulting weght loss?

No.

Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Can't anorexia and improvement in appetite be disease mediated/oriented
either for spread or for cure of disease? However loss of appetite be
may also be possible as a direction towards cure for decreasing
nutients/starving the disease causing agents in latencies/toumor?
It is thought provoking to many TB specialists when I discudssed.

Anorexia serves to inform the physician that there is a problem.

Yes, but can loss or gain of appetite indicate that whether patient is
progressing toward increase in disease or cure of disease respectively?

Loss of appetite indicates a disruption in homeostasis.

Do patients, having latent stage of TB, get episodes of acute type
symptoms of TB, when he is naturally improving by gaining immune
strength due to adopting healthful practices?

No.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld

"Alternate Identity" wrote in message
...
In article om,
"Kumar" wrote:


That's it! You are killfiled along with Chung for crossposting...

Ass hole!!!!

FWIW, kumar simply replied to Chung. It was Chung who added
rec.food.cooking.

GG

Alternate Identity wrote:
In article om,
"Kumar" wrote:


That's it! You are killfiled along with Chung for crossposting...

Ass hole!!!!

Do you want to tell anything?

Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Can it be taken: a person having predispositions to any chronic
disease(esp.those having weight loss as promient symptom eg diabetes,
TB etc.) got some symptoms of disease but not yet lost substancial and
unintentional weight, is either in early stage or not really got frank
disease and can be cured by adopting better health practices without
medicines?

Weight loss is a physical finding and not a symptom. The symptom that
leads to weight loss is anorexia which is loss of appetite.

Yes, but can't anorexia be disease mediated/oriented--either for spread
or for cure of disease?

It serves the purpose of letting a physician know that there is a
problem.

What causes anorexia?

The underlying problem that makes a person unwell.

Yes, then anorexia is underlying problem's oriented? I think most
other species than humans get anorexia, don't eat for sometimes and get
cured naturally. Is it a natural defence initiation to cure the disease
as fasting effect or to balance accumulations?

Whether getting anorexia is a direction towards progress of disease or
towards getting cure?

In short, whether weght lost symptom is must to declared patient with
frank disease in which weght loss is an important symptom?

Folks with TB lose their appetite. Folks with type 1 diabetes also
lose their appetite. Folkd with type 2 diabetes generally do not lose
their appetite and so they gain weight thereby worsening their
condition.

What about when type2 become IDDM or frank dibetic?

Then the type 2 diabetic typically starts losing his/her appetite.

It means real disease is matured only when patient start losing his/her
appetite resulting into weight loss?

No. It means there is a loss of homeostasis.

Can't loss of homeostasis happen due to maturity of disease?

Do they loose their
appetite if medicated or not medicated?

With loss of homeostasis.

Let us think about TB. Weight loss is an important symptom of active TB

They lose weight from eating less because they have lost their
appetite.

Weght loss may not be there when TB bacterias are in latent stage. I
think, there can be acute/active stage type symptoms when 1.disease is
spreading(direction of progress of disease on wekening of immunity)) 2.
when disease is occasionally controled(direction towards natural cure
by getting strong immunity). But weght loss OR no weight loss/gain
weight can be the differenciating symptom of either disease is toward
spread or toward cure. Right??

See above.

Don't bacterias kill cells resulting weght loss?

No.

Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Whether they do in initial acute stage or in latent stage or in later
active stage?

Can't anorexia and improvement in appetite be disease mediated/oriented
either for spread or for cure of disease? However loss of appetite be
may also be possible as a direction towards cure for decreasing
nutients/starving the disease causing agents in latencies/toumor?
It is thought provoking to many TB specialists when I discudssed.

Anorexia serves to inform the physician that there is a problem.

Yes, but can loss or gain of appetite indicate that whether patient is
progressing toward increase in disease or cure of disease respectively?

Loss of appetite indicates a disruption in homeostasis.

Can't disruption in homeostasis be temporary and for getting cure by
initiatation of fasting for its good effects?

Do patients, having latent stage of TB, get episodes of acute type
symptoms of TB, when he is naturally improving by gaining immune
strength due to adopting healthful practices?

No.

Can improved defence strenght kill/handle those bacterias when they are
encapsuled when in latent stage? It can be thought about cancer cells
in tumor.

"The metabolic syndrome is characterized by a group of metabolic risk
factors in one person. They include:

Central obesity (excessive fat tissue in and around the abdomen)
Atherogenic dyslipidemia (blood fat disorders - mainly high
triglycerides and low HDL cholesterol - that foster plaque buildups
in artery walls)
Insulin resistance or glucose intolerance (the body can't properly
use insulin or blood sugar)
Prothrombotic state (e.g., high fibrinogen or plasminogen activator
inhibitor [-1] in the blood)
Raised blood pressure (130/85 mmHg or higher)
Proinflammatory state (e.g., elevated high-sensitivity C-reactive
protein in the blood)
The underlying causes of this syndrome are overweight/obesity, physical
inactivity and genetic factors. People with the metabolic syndrome are
at increased risk of coronary heart disease, other diseases related to
plaque buildups in artery walls (e.g., stroke and peripheral vascular
disease) and type 2 diabetes...
The biologic mechanisms at the molecular level between insulin
resistance and metabolic risk factors aren't fully understood and
appear to be complex.

http://www.americanheart.org/present...identifier=534 "

The above link gives details of "metabolic syndromes? To look deeply,
does it indicate that increased lipid levels are mostly related to
getting most complications related to diabetes? Can we get
complications as indicated for diabetes just by hyperglycemia without
hyperlipidemia?

Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Can it be taken: a person having predispositions to any chronic
disease(esp.those having weight loss as promient symptom eg diabetes,
TB etc.) got some symptoms of disease but not yet lost substancial and
unintentional weight, is either in early stage or not really got frank
disease and can be cured by adopting better health practices without
medicines?

Weight loss is a physical finding and not a symptom. The symptom that
leads to weight loss is anorexia which is loss of appetite.

Yes, but can't anorexia be disease mediated/oriented--either for spread
or for cure of disease?

It serves the purpose of letting a physician know that there is a
problem.

What causes anorexia?

The underlying problem that makes a person unwell.

Yes, then anorexia is underlying problem's oriented?

It is pathological.

I think most
other species than humans get anorexia, don't eat for sometimes and get
cured naturally. Is it a natural defence initiation to cure the disease
as fasting effect or to balance accumulations?

No.

Whether getting anorexia is a direction towards progress of disease or
towards getting cure?

It remains simply a symptom.

In short, whether weght lost symptom is must to declared patient with
frank disease in which weght loss is an important symptom?

Folks with TB lose their appetite. Folks with type 1 diabetes also
lose their appetite. Folkd with type 2 diabetes generally do not lose
their appetite and so they gain weight thereby worsening their
condition.

What about when type2 become IDDM or frank dibetic?

Then the type 2 diabetic typically starts losing his/her appetite.

It means real disease is matured only when patient start losing his/her
appetite resulting into weight loss?

No. It means there is a loss of homeostasis.

Can't loss of homeostasis happen due to maturity of disease?

No.

Do they loose their
appetite if medicated or not medicated?

With loss of homeostasis.

Let us think about TB. Weight loss is an important symptom of active TB

They lose weight from eating less because they have lost their
appetite.

Weght loss may not be there when TB bacterias are in latent stage. I
think, there can be acute/active stage type symptoms when 1.disease is
spreading(direction of progress of disease on wekening of immunity)) 2.
when disease is occasionally controled(direction towards natural cure
by getting strong immunity). But weght loss OR no weight loss/gain
weight can be the differenciating symptom of either disease is toward
spread or toward cure. Right??

See above.

Don't bacterias kill cells resulting weght loss?

No.

Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Whether they do in initial acute stage or in latent stage or in later
active stage?

In active and not latent stages.

Can't anorexia and improvement in appetite be disease mediated/oriented
either for spread or for cure of disease? However loss of appetite be
may also be possible as a direction towards cure for decreasing
nutients/starving the disease causing agents in latencies/toumor?
It is thought provoking to many TB specialists when I discudssed.

Anorexia serves to inform the physician that there is a problem.

Yes, but can loss or gain of appetite indicate that whether patient is
progressing toward increase in disease or cure of disease respectively?

Loss of appetite indicates a disruption in homeostasis.

Can't disruption in homeostasis be temporary and for getting cure by
initiatation of fasting for its good effects?

No.

Do patients, having latent stage of TB, get episodes of acute type
symptoms of TB, when he is naturally improving by gaining immune
strength due to adopting healthful practices?

No.

Can improved defence strenght kill/handle those bacterias when they are
encapsuled when in latent stage? It can be thought about cancer cells
in tumor.

No.

"The metabolic syndrome is characterized by a group of metabolic risk
factors in one person. They include:

Central obesity (excessive fat tissue in and around the abdomen)
Atherogenic dyslipidemia (blood fat disorders - mainly high
triglycerides and low HDL cholesterol - that foster plaque buildups
in artery walls)
Insulin resistance or glucose intolerance (the body can't properly
use insulin or blood sugar)
Prothrombotic state (e.g., high fibrinogen or plasminogen activator
inhibitor [-1] in the blood)
Raised blood pressure (130/85 mmHg or higher)
Proinflammatory state (e.g., elevated high-sensitivity C-reactive
protein in the blood)
The underlying causes of this syndrome are overweight/obesity, physical
inactivity and genetic factors. People with the metabolic syndrome are
at increased risk of coronary heart disease, other diseases related to
plaque buildups in artery walls (e.g., stroke and peripheral vascular
disease) and type 2 diabetes...
The biologic mechanisms at the molecular level between insulin
resistance and metabolic risk factors aren't fully understood and
appear to be complex.

http://www.americanheart.org/present...identifier=534 "

The above link gives details of "metabolic syndromes? To look deeply,
does it indicate that increased lipid levels are mostly related to
getting most complications related to diabetes?

No.

Can we get
complications as indicated for diabetes just by hyperglycemia without
hyperlipidemia?

Yes.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld

Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Whether they do in initial acute stage or in latent stage or in later
active stage?

In active and not latent stages.


Whether body's defence mechanism mediate any change in appetite or in
absorption of any bio-substance (say iron) during latent stage? Can
latencies be treated as persisting infection alike persistent
inflammation?

Can defence responses kill/handle those bacterias when they are
encapsuled when in latent stage? It can be thought about cancer cells
in tumor.

No.

Can defence responses mediate to bring the bacterias out of
encapsuled/latent state unabling to remove or kill them?

"The metabolic syndrome is characterized by a group of metabolic risk
factors in one person. They include:

Central obesity (excessive fat tissue in and around the abdomen)
Atherogenic dyslipidemia (blood fat disorders - mainly high
triglycerides and low HDL cholesterol - that foster plaque buildups
in artery walls)
Insulin resistance or glucose intolerance (the body can't properly
use insulin or blood sugar)
Prothrombotic state (e.g., high fibrinogen or plasminogen activator
inhibitor [-1] in the blood)
Raised blood pressure (130/85 mmHg or higher)
Proinflammatory state (e.g., elevated high-sensitivity C-reactive
protein in the blood)
The underlying causes of this syndrome are overweight/obesity, physical
inactivity and genetic factors. People with the metabolic syndrome are
at increased risk of coronary heart disease, other diseases related to
plaque buildups in artery walls (e.g., stroke and peripheral vascular
disease) and type 2 diabetes...
The biologic mechanisms at the molecular level between insulin
resistance and metabolic risk factors aren't fully understood and
appear to be complex.

http://www.americanheart.org/present...identifier=534 "

The above link gives details of "metabolic syndromes? To look deeply,
does it indicate that increased lipid levels are mostly related to
getting most complications related to diabetes?

No.

Can we get
complications as indicated for diabetes just by hyperglycemia without
hyperlipidemia?

Yes.

Which is **more** responsible for getting complications related to
diabetes--elevated blood gulucose level or elevated lipids level?

Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Whether they do in initial acute stage or in latent stage or in later
active stage?

In active and not latent stages.

Whether body's defence mechanism mediate any change in appetite or in
absorption of any bio-substance (say iron) during latent stage?

The immune system is involved in disrupting homeostasis leading to
anorexia.

Can
latencies be treated as persisting infection alike persistent
inflammation?

No.

Can defence responses kill/handle those bacterias when they are
encapsuled when in latent stage? It can be thought about cancer cells
in tumor.

No.

Can defence responses mediate to bring the bacterias out of
encapsuled/latent state unabling to remove or kill them?

No.

"The metabolic syndrome is characterized by a group of metabolic risk
factors in one person. They include:

Central obesity (excessive fat tissue in and around the abdomen)
Atherogenic dyslipidemia (blood fat disorders - mainly high
triglycerides and low HDL cholesterol - that foster plaque buildups
in artery walls)
Insulin resistance or glucose intolerance (the body can't properly
use insulin or blood sugar)
Prothrombotic state (e.g., high fibrinogen or plasminogen activator
inhibitor [-1] in the blood)
Raised blood pressure (130/85 mmHg or higher)
Proinflammatory state (e.g., elevated high-sensitivity C-reactive
protein in the blood)
The underlying causes of this syndrome are overweight/obesity, physical
inactivity and genetic factors. People with the metabolic syndrome are
at increased risk of coronary heart disease, other diseases related to
plaque buildups in artery walls (e.g., stroke and peripheral vascular
disease) and type 2 diabetes...
The biologic mechanisms at the molecular level between insulin
resistance and metabolic risk factors aren't fully understood and
appear to be complex.

http://www.americanheart.org/present...identifier=534 "

The above link gives details of "metabolic syndromes? To look deeply,
does it indicate that increased lipid levels are mostly related to
getting most complications related to diabetes?

No.

Can we get
complications as indicated for diabetes just by hyperglycemia without
hyperlipidemia?

Yes.

Which is **more** responsible for getting complications related to
diabetes--elevated blood gulucose level or elevated lipids level?

Depends on the individual. This will vary from person to person.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld

Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Whether they do in initial acute stage or in latent stage or in later
active stage?

In active and not latent stages.

Whether body's defence mechanism mediate any change in appetite or in
absorption of any bio-substance (say iron) during latent stage?

The immune system is involved in disrupting homeostasis leading to
anorexia.

Can
latencies be treated as persisting infection alike persistent
inflammation?

No.

Can defence responses kill/handle those bacterias when they are
encapsuled when in latent stage? It can be thought about cancer cells
in tumor.

No.

Can defence responses mediate to bring the bacterias out of
encapsuled/latent state unabling to remove or kill them?

No.

"The metabolic syndrome is characterized by a group of metabolic risk
factors in one person. They include:

Central obesity (excessive fat tissue in and around the abdomen)
Atherogenic dyslipidemia (blood fat disorders - mainly high
triglycerides and low HDL cholesterol - that foster plaque buildups
in artery walls)
Insulin resistance or glucose intolerance (the body can't properly
use insulin or blood sugar)
Prothrombotic state (e.g., high fibrinogen or plasminogen activator
inhibitor [-1] in the blood)
Raised blood pressure (130/85 mmHg or higher)
Proinflammatory state (e.g., elevated high-sensitivity C-reactive
protein in the blood)
The underlying causes of this syndrome are overweight/obesity, physical
inactivity and genetic factors. People with the metabolic syndrome are
at increased risk of coronary heart disease, other diseases related to
plaque buildups in artery walls (e.g., stroke and peripheral vascular
disease) and type 2 diabetes...
The biologic mechanisms at the molecular level between insulin
resistance and metabolic risk factors aren't fully understood and
appear to be complex.

http://www.americanheart.org/present...identifier=534 "

The above link gives details of "metabolic syndromes? To look deeply,
does it indicate that increased lipid levels are mostly related to
getting most complications related to diabetes?

No.

Can we get
complications as indicated for diabetes just by hyperglycemia without
hyperlipidemia?

Yes.

Which is **more** responsible for getting complications related to
diabetes--elevated blood gulucose level or elevated lipids level?

Depends on the individual. This will vary from person to person.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld
I think My above questions are mostly settled. It looked these were
exchanged and mostly settled by the grace & love of LORD, who is same
for you and me. Many thanks. Few more to conclude:-

Is there any relation between sulphur with diabetes?
Don't sulphur in our body, exist in free form but only bounded in
protiens? Can't we get sulphur imbalances in our bodies?

What are the effects of levels of gastric acid, bile and pancretic
bicarbonate on each other? Can any unstability in any of these effect
level or effectiveness of other?

..

Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Whther TB bacterias do such changes in homoestatis which can be a cause
of loss of appetite resulting into loss of weight?

They do.

Whether they do in initial acute stage or in latent stage or in later
active stage?

In active and not latent stages.

Whether body's defence mechanism mediate any change in appetite or in
absorption of any bio-substance (say iron) during latent stage?

The immune system is involved in disrupting homeostasis leading to
anorexia.

Can
latencies be treated as persisting infection alike persistent
inflammation?

No.

Can defence responses kill/handle those bacterias when they are
encapsuled when in latent stage? It can be thought about cancer cells
in tumor.

No.

Can defence responses mediate to bring the bacterias out of
encapsuled/latent state unabling to remove or kill them?

No.

"The metabolic syndrome is characterized by a group of metabolic risk
factors in one person. They include:

Central obesity (excessive fat tissue in and around the abdomen)
Atherogenic dyslipidemia (blood fat disorders - mainly high
triglycerides and low HDL cholesterol - that foster plaque buildups
in artery walls)
Insulin resistance or glucose intolerance (the body can't properly
use insulin or blood sugar)
Prothrombotic state (e.g., high fibrinogen or plasminogen activator
inhibitor [-1] in the blood)
Raised blood pressure (130/85 mmHg or higher)
Proinflammatory state (e.g., elevated high-sensitivity C-reactive
protein in the blood)
The underlying causes of this syndrome are overweight/obesity, physical
inactivity and genetic factors. People with the metabolic syndrome are
at increased risk of coronary heart disease, other diseases related to
plaque buildups in artery walls (e.g., stroke and peripheral vascular
disease) and type 2 diabetes...
The biologic mechanisms at the molecular level between insulin
resistance and metabolic risk factors aren't fully understood and
appear to be complex.

http://www.americanheart.org/present...identifier=534 "

The above link gives details of "metabolic syndromes? To look deeply,
does it indicate that increased lipid levels are mostly related to
getting most complications related to diabetes?

No.

Can we get
complications as indicated for diabetes just by hyperglycemia without
hyperlipidemia?

Yes.

Which is **more** responsible for getting complications related to
diabetes--elevated blood gulucose level or elevated lipids level?

Depends on the individual. This will vary from person to person.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld

I think My above questions are mostly settled. It looked these were
exchanged and mostly settled by the grace & love of LORD, who is same
for you and me. Many thanks. Few more to conclude:-

Is there any relation between sulphur with diabetes?

No.

Don't sulphur in our body, exist in free form but only bounded in
protiens?

In proteins.

Can't we get sulphur imbalances in our bodies?

No.

What are the effects of levels of gastric acid, bile and pancretic
bicarbonate on each other? Can any unstability in any of these effect
level or effectiveness of other?

Not in practice.

LORD willing, will be available to "glow" and chat about this and other
things like cardiology, diabetes, cooking and nutrition that interest
those following this thread here during the next on-line chat now
(02/09/06):

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me:

http://tinyurl.com/bgfqt

Prayerfully in Christ's love,

Andrew
http://tinyurl.com/8juld

Andrew B. Chung, MD/PhD wrote:

I think My above questions are mostly settled. It looked these were
exchanged and mostly settled by the grace & love of LORD, who is same
for you and me. Many thanks. Few more to conclude:-

Is there any relation between sulphur with diabetes?

No.

Insulin and most diabetic medicines are sulphur based. How such sulphur
is catabolized?

Don't sulphur in our body, exist in free form but only bounded in
protiens?

In proteins.

On intake and absorption of sulphur, how it is metabolized?

Can't we get sulphur imbalances in our bodies?

No.

What happens on excessive intake of sulphur or sulphur containing
foods?

What are the effects of levels of gastric acid, bile and pancretic
bicarbonate on each other? Can any unstability in any of these effect
level or effectiveness of other?

Not in practice.

What is the effect of lesser or higher bile secretions?

Whether bile is only secreted on fats presence?

On Sun, 5 Feb 2006 11:05:42 -0800, "GaryG"
Huffed and Puffed the following into the madness of usenet:

"Alternate Identity" wrote in message
...
In article om,
"Kumar" wrote:


That's it! You are killfiled along with Chung for crossposting...

Ass hole!!!!

FWIW, kumar simply replied to Chung. It was Chung who added
rec.food.cooking.

GG



actually Kumar has been starting threads and intentionally cross
posting them to chungs favorite hit list.

--
Mâck©® Deltec CoZmore Pumper
Type 1 since 1975
http://www.alt-support-diabetes.org
http://www.diabetic-talk.org
http://www.insulin-pumpers.org



"To announce that there must be no criticism of the
President, or that we are to stand by the President
right or wrong, is not only unpatriotic and servile,
but is morally treasonable to the American public."
....Theodore Roosevelt

(o o)
--ooO-(_)-Ooo--------------------

"I don't know half of you
half as well as I should like;
and I like less than half of you
half as well as you deserve."



Jesus never hated anyone.

Kumar wrote:
Andrew B. Chung, MD/PhD wrote:

I think My above questions are mostly settled. It looked these were
exchanged and mostly settled by the grace & love of LORD, who is same
for you and me. Many thanks. Few more to conclude:-

Is there any relation between sulphur with diabetes?

No.

Insulin and most diabetic medicines are sulphur based.

Actually, they are not really sulphur based.

How such sulphur
is catabolized?

Sulphur is an element. It can not be catabolized.

Don't sulphur in our body, exist in free form but only bounded in
protiens?

In proteins.

On intake and absorption of sulphur, how it is metabolized?

It is an element. It can not be metabolized.

Can't we get sulphur imbalances in our bodies?

No.

What happens on excessive intake of sulphur or sulphur containing
foods?

Excessive sulphur intake is lethal.

What are the effects of levels of gastric acid, bile and pancretic
bicarbonate on each other? Can any unstability in any of these effect
level or effectiveness of other?

Not in practice.

What is the effect of lesser or higher bile secretions?

A fatty meal stimulates increased bile secretion to aid in its
digestion.

Whether bile is only secreted on fats presence?

Largely.

Will be available to "glow" and chat about this and other things like
cardiology, diabetes, cooking and nutrition that interest those
following this thread here during the next on-line chat (02/09/06) from
6 to 7 pm EST:

http://tinyurl.com/cpayh

For those who are put off by the signature, my advance apologies for
how the LORD has reshaped me: